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Since several ATGs and lysosomal cathepsin proteases depend on cysteines for their activities they can be the sensors or transducers of changes in the intracellular redox state through mechanisms involving redox signaling

226 Cu deficiency can also alter the expression of myocardial gene profile, for example by upregulating the inflammatory cytokine TNF- and genes involved in regulating cardiac contractility, fibrosis, and calcium cycling, all of which may underlie the changes in cardiac function under Cu-deficient conditions

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Based on these findings, Xinyu Zhou et al